Translational suppression by Ca2+ ionophores: Reversibility and roles of Ca2+ mobilization, Ca2+ influx, and nucleotide depletion-Reference-Cited by-同舟云学术

Translational suppression by Ca2+ ionophores: Reversibility and roles of Ca2+ mobilization, Ca2+ influx, and nucleotide depletion

Published:1996-04 Issue:2 Volume:12 Page:101-113
ISSN:0742-2091
Container-title:Cell Biology and Toxicology
language:en
Short-container-title:Cell Biol Toxicol

Author:

Gmitter D.,Brostrom C.O.,Brostrom M.A.

Publisher

Springer Science and Business Media LLC

Subject

Health, Toxicology and Mutagenesis,Cell Biology,Toxicology

Link

http://link.springer.com/content/pdf/10.1007/BF00143360.pdf

Reference30 articles.

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2. Albert PR, Tashjian AH Jr. Ionomycin acts as an ionophore to release TRH-regulated Ca2+ stores from GH4C1 cells. Am J Physiol. 1986;251:C887?91.

3. Bartlett JD, Luethy JD, Carlson SG, Sollott SJ, Holbrook NJ. Calcium ionophore A-23187 induces expression of the growth arrest and DNA damage inducible CCAAT/enhancer-binding protein (C/EBP)-related gene, gadd 153- Ca2+ increases transcriptional activity and messenger RNA stability. J Biol Chem. 1992;267:20465?70.

4. Beccari JD, Datti A, Orlacchio A, Farinelli S, Blasi E. Calcium ionophore A-23187 inhibits the secretion of beta-hexosaminidase from the GG2EE mouse macrophage line. Biochem Int. 1992;27:783?91.

5. Bernardi P, Angrilli A, Ambrosi V, Azzone GF. Activation of latent K+ uniport in mitochondria treated with the ionophore A23187. J Biol Chem. 1989;264:18902?6.

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