Nitric oxide, but not vasopressin V 2 receptor-mediated vasodilation, modulates vasopressin-induced renal vasoconstriction in rats-Reference-Cited by-同舟云学术

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Nitric oxide, but not vasopressin V 2 receptor-mediated vasodilation, modulates vasopressin-induced renal vasoconstriction in rats

Published:2000-02-07 Issue:3 Volume:361 Page:319-326
ISSN:0028-1298
Container-title:Naunyn-Schmiedeberg's Archives of Pharmacology
language:
Short-container-title:Naunyn-Schmiedeberg's Archives of Pharmacology

Author:

Loichot C.,Cazaubon C.,De Jong W.,Helwig J.-J.,Nisato D.,Imbs J.-L.,Barthelmebs M.

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology,General Medicine

Link

http://link.springer.com/content/pdf/10.1007/s002109900187.pdf

Cited by 7 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Renal vasoconstriction by vasopressin V1a receptors is modulated by nitric oxide, prostanoids, and superoxide but not the ADP ribosyl cyclase CD38;American Journal of Physiology-Renal Physiology;2014-05-15

2. Sustained Elevated Levels of Circulating Vasopressin Selectively Stimulate the Proliferation of Kidney Tubular Cells via the Activation of V2 Receptors;Endocrinology;2008-09-11

3. Effects of Novel Vasopressin Receptor Antagonists on Renal Function and Cardiac Hypertrophy in Rats with Experimental Congestive Heart Failure;Journal of Pharmacology and Experimental Therapeutics;2008-05-08

4. Future Pharmacologic Agents for Treatment of Heart Failure in Children;Pediatric Cardiology;2006-08-23

5. Systemic and renal macro- and microcirculatory responses to arginine vasopressin in endotoxic rabbits*;Critical Care Medicine;2004-09

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