Dimethyl Fumarate Inhibits Fibroblast Like Synoviocytes-mediated Inflammation and Joint Destruction in Rheumatoid Arthritis
Author:
Funder
Mazandaran University of Medical Sciences
Publisher
Springer Science and Business Media LLC
Subject
Immunology,Immunology and Allergy
Link
https://link.springer.com/content/pdf/10.1007/s10753-022-01759-1.pdf
Reference34 articles.
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2. Zafari, P., K. Yari, S. Mostafaei, N. Iranshahi, S. Assar, A. Fekri, et al. 2018. Analysis of Helios gene expression and Foxp3 TSDR methylation in the newly diagnosed Rheumatoid Arthritis patients. Immunological Investigations 47 (6): 632–642.
3. Lefevre, S., F. Meier, E. Neumann, and U. Muller-Ladner. 2015. Role of synovial fibroblasts in rheumatoid arthritis. Current Pharmaceutical Design. 21 (2): 130–41.
4. Hillen, J., C. Geyer, M. Heitzmann, D. Beckmann, A. Krause, and I. Winkler, et al. 2017. Structural cartilage damage attracts circulating rheumatoid arthritis synovial fibroblasts into affected joints. Arthritis Research & Therapy. 19 (1): 1–11.
5. Samimi, Z., B. Kardideh, P. Zafari, F. Bahrehmand, S.A. Roghani, and M. Taghadosi. 2019. The impaired gene expression of adenosine monophosphate-activated kinase (AMPK), a key metabolic enzyme in leukocytes of newly diagnosed rheumatoid arthritis patients. Molecular Biology Reports 46 (6): 6353–6360.
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