Anti-inflammatory Action of Metformin with Respect to CX3CL1/CX3CR1 Signaling in Human Placental Circulation in Normal-Glucose Versus High-Glucose Environments
Author:
Funder
Medical University of Warsaw
Publisher
Springer Science and Business Media LLC
Subject
Immunology,Immunology and Allergy
Link
http://link.springer.com/article/10.1007/s10753-018-0867-7/fulltext.html
Reference81 articles.
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2. Desoye, G., and S. Hauguel-de Mouzon. 2007. The human placenta in gestational diabetes mellitus. The insulin and cytokine network. Diabetes Care 30 (Suppl 2): S120–S126 Erratum in: Diabetes Care 2007; 30: 3154.
3. Navarro-González, J.F., and C. Mora-Fernández. 2011. Inflammatory pathways. In Diabetes and the kidney. Contributions to nephrology, ed. K.N. Lai and S.C.W. Tang, vol. 170, 113–123. Basel: Karger.
4. Dragomir, E., I. Manduteanu, M. Calin, A.M. Gan, D. Stan, R.R. Koenen, et al. 2008. High glucose conditions induce upregulation of fractalkine and monocyte chemotactic protein-1 in human smooth muscle cells. Thrombosis and Haemostasis 100: 1155–1165.
5. Szukiewicz, D., J. Kochanowski, M. Pyzlak, G. Szewczyk, A. Stangret, and T.K. Mittal. 2013. Fractalkine (CX3CL1) and its receptor CX3CR1 may contribute to increased angiogenesis in diabetic placenta. Mediators of Inflammation 2013: 437576.
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