Increased expression of TCF3, transcription factor 3, is a defense response against methylmercury toxicity in mouse neuronal C17.2 cells
Author:
Funder
Japan Society for the Promotion of Science
Publisher
Springer Science and Business Media LLC
Subject
Health, Toxicology and Mutagenesis,Toxicology
Link
https://link.springer.com/content/pdf/10.1007/s43188-021-00087-0.pdf
Reference24 articles.
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2. Simmons-Willis TA, Koh AS, Clarkson TW, Ballatori N (2002) Transport of a neurotoxicant by molecular mimicry: the methylmercury-l-cysteine complex is a substrate for human l-type large neutral amino acid transporter (LAT) 1 and LAT2. Biochem J 367:239–246. https://doi.org/10.1042/BJ20020841
3. Harada M (1995) Minamata disease: methylmercury poisoning in Japan caused by environmental pollution. Crit Rev Toxicol 25:1–24. https://doi.org/10.3109/10408449509089885
4. Harada M (1978) Congenital minamata disease: intrauterine methylmercury poisoning. Teratology 18:285–288. https://doi.org/10.1002/tera.1420180216
5. Unoki T, Abiko Y, Toyama T, Uehara T, Tsuboi K, Nishida M, Kaji T, Kumagai Y (2016) Methylmercury, an environmental electrophile capable of activation and disruption of the Akt/CREB/Bcl-2 signal transduction pathway in SH-SY5Y cells. Sci Rep 6:28944. https://doi.org/10.1038/srep28944
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1. Methylmercury Induces Apoptosis in Mouse C17.2 Neural Stem Cells through the Induction of OSGIN1 Expression by NRF2;International Journal of Molecular Sciences;2024-03-30
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