Detection of latent thymidine kinase-deficient herpes simplex virus in trigeminal ganglia of mice using the polymerase chain reaction
Author:
Publisher
Springer Science and Business Media LLC
Subject
Virology,General Medicine
Link
http://link.springer.com/content/pdf/10.1007/BF01318359.pdf
Reference20 articles.
1. Coen DM, Kosz-Vnenchak M, Jacobson JG, Leib DA, Bogard CL, Schaffer PA, Tyler KL, Knipe DM (1989) Thymidine kinase-negative herpes simplex virus mutants establish latency in mouse trigeminal ganglia but do not reactivate. Proc Natl Acad Sci USA 86: 4736?4740
2. Darby G, Field HJ, Salisbury SA (1981) Altered substrate specificity of herpes simplex virus thymidine kinase confers acyclovir-resistance. Nature 289: 81?83
3. Efstathiou S, Kemp S, Darby G, Minson RC (1989) The role of herpes simplex virus type 1 thymidine kinase in pathogenesis. J Gen Virol 70: 869?879
4. Field HJ, Wildy P (1978) The pathogenicity of thymidine-kinase-deficient mutants of herpes simplex virus in mice. J Hyg (Cambridge) 81: 267?277
5. Gordon YJ, Rock DL (1984) Cocultivation versus blot hybridzation for the detection of trigeminal ganglionic latency following corneal inoculation with HSV-1 strains of varying TK expression and pathogenicity. Curr Eye Res 3: 1097?1100
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