Co-cultivation versus blot hybridization for the detection of trigeminal ganglionic latency following corneal inoculation with HSV-1 strains of varying TK expression and pathogenicity
Author:
Publisher
Informa UK Limited
Subject
Cellular and Molecular Neuroscience,Sensory Systems,Ophthalmology
Link
http://www.tandfonline.com/doi/pdf/10.3109/02713688409000807
Reference14 articles.
1. Recovery of Herpes-Simplex Virus from Human Trigeminal Ganglions
2. Latent Herpes Simplex Virus from Trigeminal Ganglia of Rabbits with Recurrent Eye Infection
3. Recurrence Phenotypes and Establishment of Latency Following Rabbit Keratitis Produced by Multiple Herpes Simplex Virus Strains
4. Herpes simplex virus DNA sequences in the CNS of latently infected mice
5. Detection of herpes simplex virus mRNA in latently infected trigeminal ganglion neurons by in situ hybridization
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1. Role of Herpes Simplex Virus Type 1 (HSV-1) Glycoprotein K (gK) Pathogenic CD8+ T Cells in Exacerbation of Eye Disease;Frontiers in Immunology;2018-12-07
2. A Recombinant Herpes Simplex Virus Type 1 Expressing Two Additional Copies of gK Is More Pathogenic than Wild-Type Virus in Two Different Strains of Mice;Journal of Virology;2007-12
3. Improved Protection from Primary Ocular HSV-1 Infection and Establishment of Latency Using Multigenic DNA Vaccines;Investigative Opthalmology & Visual Science;2004-02-01
4. Either a CD4+or CD8+T cell function is sufficient for clearance of infectious virus from trigeminal ganglia and establishment of herpes simplex virus type 1 latency in mice;Microbial Pathogenesis;1999-12
5. A 371-nucleotide region between the herpes simplex virus type 1 (HSV-1) LAT promoter and the 2-kilobase LAT is not essential for efficient spontaneous reactivation of latent HSV-1;Journal of Virology;1996-03
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