Long non-coding RNAs TUG1 and MEG3 in patients with type 2 diabetes and their association with endoplasmic reticulum stress markers
Author:
Funder
Iran University of Medical Sciences
Publisher
Springer Science and Business Media LLC
Subject
Endocrinology,Endocrinology, Diabetes and Metabolism
Link
https://link.springer.com/content/pdf/10.1007/s40618-023-02007-5.pdf
Reference41 articles.
1. Surampudi PN, John-Kalarickal J, Fonseca VA (2009) Emerging concepts in the pathophysiology of type 2 diabetes mellitus. Mount Sinai J Med J Trans Personal Med. 76(3):216–226
2. Zaccardi FWD, Yates T, Davies MJ (2016) Pathophysiology of type 1 and type 2 diabetes mellitus: a 90 year perspective. Postgrad Med J 92(1084):63–69
3. Back SH, Randal KJ (2012) Endoplasmic reticulum stress and type 2 diabetes. Ann Rev Biochem. 81:767–793
4. Saeedi P, Petersohn I, Salpea P, Malanda B, Karuranga S, Unwin N et al (2019) Global and regional diabetes prevalence estimates for 2019 and projections for 2030 and 2045: results from the International diabetes federation diabetes Atlas. Diabet Res Clin Pract. 157:107843
5. Shen H, Ming Y, Xu C, Xu Y, Zhao S, Zhang Q (2019) Deregulation of long noncoding RNA (TUG1) contributes to excessive podocytes apoptosis by activating endoplasmic reticulum stress in the development of diabetic nephropathy. J Cell Physiol 234(9):15123–15133
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