Deregulation of long noncoding RNA (TUG1) contributes to excessive podocytes apoptosis by activating endoplasmic reticulum stress in the development of diabetic nephropathy-Reference-Cited by-同舟云学术

Deregulation of long noncoding RNA (TUG1) contributes to excessive podocytes apoptosis by activating endoplasmic reticulum stress in the development of diabetic nephropathy

Published:2019-01-22 Issue:9 Volume:234 Page:15123-15133
ISSN:0021-9541
Container-title:Journal of Cellular Physiology
language:en
Short-container-title:J Cell Physiol

Author:

Shen Hongchun¹,Ming Yao¹,Xu Chuanlan¹,Xu Yanwen¹,Zhao Sha¹,Zhang Qiong¹^ORCID

Affiliation:

1. Department of Nephrology The Affiliated Traditional Medicine Hospital of Southwest Medical University Luzhou Sichuan China

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.28153

Reference38 articles.

1. The long non-coding RNA Gomafu is acutely regulated in response to neuronal activation and involved in schizophrenia-associated alternative splicing

2. Drug Discovery for Diabetic Nephropathy: Trying the Leap From Mouse to Man

3. AKT2 is essential to maintain podocyte viability and function during chronic kidney disease

4. Role of endoplasmic reticulum stress in apoptosis of differentiated mouse podocytes induced by high glucose

5. Decoding the function of nuclear long non-coding RNAs

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