Author:
Carter James E.,Odumosu Oludare,Langridge William H. R.
Publisher
Springer Science and Business Media LLC
Subject
Molecular Biology,Applied Microbiology and Biotechnology,Biochemistry,Bioengineering,Biotechnology
Reference51 articles.
1. Faria, A. M., & Weiner, H. L. (2006). Oral tolerance: Therapeutic implications for autoimmune diseases. Clinical and Developmental Immunology, 13(2–4), 143–157.
2. Hancock, W. W., Polanski, M., Zhang, J., Blogg, N., & Weiner, H. L. (1995). Suppression of insulitis in non-obese diabetic (NOD) mice by oral insulin administration is associated with selective expression of interleukin-4 and -10, transforming growth factor-beta, and prostaglandin-E. American Journal of Pathology, 147(5), 1193–1199.
3. Maron, R., Guerau-de-Arellano, M., Zhang, X., & Weiner, H. L. (2001). Oral administration of insulin to neonates suppresses spontaneous and cyclophosphamide induced diabetes in the NOD mouse. Journal of Autoimmunity, 16(1), 21–28.
4. Maron, R., Melican, N. S., & Weiner, H. L. (1999). Regulatory Th2-type T cell lines against insulin and GAD peptides derived from orally- and nasally-treated NOD mice suppress diabetes. Journal of Autoimmunity, 12(4), 251–258.
5. Polanski, M., Melican, N. S., Zhang, J., & Weiner, H. L. (1997). Oral administration of the immunodominant B-chain of insulin reduces diabetes in a co-transfer model of diabetes in the NOD mouse and is associated with a switch from Th1 to Th2 cytokines. Journal of Autoimmunity, 10(4), 339–346.
Cited by
14 articles.
订阅此论文施引文献
订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献