Correlations between age, functional status, and the senescence-associated proteins HMGB2 and p16INK4a
Author:
Funder
Commonwealth of Pennsylvania
Publisher
Springer Science and Business Media LLC
Subject
Geriatrics and Gerontology,Ageing
Link
http://link.springer.com/article/10.1007/s11357-018-0015-1/fulltext.html
Reference17 articles.
1. Aird KM, Iwasaki O, Kossenkov AV, Tanizawa H, Fatkhutdinov N, Bitler BG, le L, Alicea G, Yang TL, Johnson FB, Noma KI, Zhang R (2016) HMGB2 orchestrates the chromatin landscape of senescence-associated secretory phenotype gene loci. J Cell Biol 215(3):325–334
2. Baker DJ, Wijshake T, Tchkonia T, LeBrasseur NK, Childs BG, van de Sluis B, Kirkland JL, van Deursen JM (2011) Clearance of p16Ink4a-positive senescent cells delays ageing-associated disorders. Nature 479(7372):232–236
3. Bianchi ME, Agresti A (2005) HMG proteins: dynamic players in gene regulation and differentiation. Curr Opin Genet Dev 15(5):496–506
4. Davalos AR, Kawahara M, Malhotra GK, Schaum N, Huang J, Ved U, Beausejour CM, Coppe JP, Rodier F, Campisi J (2013) p53-dependent release of Alarmin HMGB1 is a central mediator of senescent phenotypes. J Cell Biol 201(4):613–629
5. He S, Sharpless NE (2017) Senescence in health and disease. Cell 169(6):1000–1011
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