Cellular Filamentation After Sublethal High-Pressure Shock in Escherichia coli K12 is Mrr Dependent
Author:
Publisher
Springer Science and Business Media LLC
Subject
Applied Microbiology and Biotechnology,General Medicine,Microbiology
Link
http://link.springer.com/content/pdf/10.1007/s00284-013-0449-8.pdf
Reference16 articles.
1. Abe A, Furukawa S, Migita Y, Tanaka M, Ogihara H, Morinaga Y (2013) Sublethal high hydrostatic pressure treatment reveals the importance of genes coding cytoskeletal protein in Escherichia coli morphogenesis. Curr Microbiol (in press)
2. Kawarai T, Wachi M, Ogino H, Furukawa S, Suzuki K, Ogihara H, Yamasaki M (2004) SulA-independent filamentation of Escherichia coli during growth after release from high hydrostatic pressure treatment. Appl Microbiol Biotechnol 64:255–262
3. Aertsen A, Van Houdt R, Vanoirbeek K, Michiels CW (2004) An SOS response induced by high pressure in Escherichia coli. J Bacteriol 186:6133–6141
4. Aertsen A, Faster D, Michiels CW (2005) Induction of shiga toxin-converting prophage in Escherichia coli by high hydrostatic pressure. Appl Environ Microbiol 71:1155–1162
5. Aertsen A, Michiels CW (2005) Mrr instigates the SOS response after high pressure stress in Escherichia coli. Mol Microbiol 58:1381–1391
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3. High pressure activation of the Mrr restriction endonuclease in Escherichia coli involves tetramer dissociation;Nucleic Acids Research;2017-03-21
4. Cellular localization and dynamics of the Mrr type IV restriction endonuclease of Escherichia coli;Nucleic Acids Research;2014-01-13
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