The lack of functional nicotinamide nucleotide transhydrogenase only moderately contributes to the impairment of glucose tolerance and glucose-stimulated insulin secretion in C57BL/6J vs C57BL/6N mice-Reference-Cited by-同舟云学术

The lack of functional nicotinamide nucleotide transhydrogenase only moderately contributes to the impairment of glucose tolerance and glucose-stimulated insulin secretion in C57BL/6J vs C57BL/6N mice

Published:2021-08-27 Issue:11 Volume:64 Page:2550-2561
ISSN:0012-186X
Container-title:Diabetologia
language:en
Short-container-title:Diabetologia

Author:

Close Anne-Françoise,Chae Heeyoung^ORCID,Jonas Jean-Christophe^ORCID

Funder

Communauté française de Belgique, Action de Recherche Concertée

Société Francophone du Diabète

Fonds De La Recherche Scientifique - FNRS

Publisher

Springer Science and Business Media LLC

Subject

Endocrinology, Diabetes and Metabolism,Internal Medicine

Link

https://link.springer.com/content/pdf/10.1007/s00125-021-05548-7.pdf

Reference48 articles.

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3. Vistisen D, Witte DR, Tabak AG et al (2014) Patterns of obesity development before the diagnosis of type 2 diabetes: the Whitehall II cohort study. PLoS Med 11(2):e1001602. https://doi.org/10.1371/journal.pmed.1001602

4. Taylor R, Al-Mrabeh A, Zhyzhneuskaya S et al (2018) Remission of Human Type 2 Diabetes Requires Decrease in Liver and Pancreas Fat Content but Is Dependent upon Capacity for β Cell Recovery. Cell Metab 28(4): 547–556; e541-e543. https://doi.org/10.1016/j.cmet.2018.07.003

5. Prentki M, Matschinsky FM, Madiraju SR (2013) Metabolic signaling in fuel-induced insulin secretion. Cell Metab 18(2):162–185. https://doi.org/10.1016/j.cmet.2013.05.018

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