Aggravation of diabetic nephropathy in BCL-2 interacting cell death suppressor (BIS)-haploinsufficient mice together with impaired induction of superoxide dismutase (SOD) activity-Reference-Cited by-同舟云学术

Aggravation of diabetic nephropathy in BCL-2 interacting cell death suppressor (BIS)-haploinsufficient mice together with impaired induction of superoxide dismutase (SOD) activity

Published:2013-09-29 Issue:1 Volume:57 Page:214-223
ISSN:0012-186X
Container-title:Diabetologia
language:en
Short-container-title:Diabetologia

Author:

Lim Ji Hee,Youn Dong-Ye,Yoo Hyung Jae,Yoon Hye Hyeon,Kim Min Young,Chung Sungjin,Kim Yong-Soo,Chang Yoon Sik,Park Cheol Whee,Lee Jeong-Hwa

Publisher

Springer Science and Business Media LLC

Subject

Endocrinology, Diabetes and Metabolism,Internal Medicine

Link

http://link.springer.com/content/pdf/10.1007/s00125-013-3064-0.pdf

Reference40 articles.

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4. Higgins DF, Kimura K, Bernhardt WM et al (2007) Hypoxia promotes fibrogenesis in vivo via HIF-1 stimulation of epithelial-to-mesenchymal transition. J Clin Invest 117:3810–3820

5. Neusser MA, Lindenmeyer MT, Moll AG et al (2010) Human nephrosclerosis triggers a hypoxia-related glomerulopathy. Am J Pathol 176:594–607

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