Presumption of innocence for beta cells: why are they vulnerable autoimmune targets in type 1 diabetes?-Reference-Cited by-同舟云学术

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Presumption of innocence for beta cells: why are they vulnerable autoimmune targets in type 1 diabetes?

Published:2020-09-07 Issue:10 Volume:63 Page:1999-2006
ISSN:0012-186X
Container-title:Diabetologia
language:en
Short-container-title:Diabetologia

Author:

Mallone Roberto^ORCID,Eizirik Decio L.^ORCID

Funder

The Leona M. and Harry B. Helmsley Charitable Trust

Fondation Francophone pour la Recherche sur le Diabète

JDRF

Fondation pour la Recherche Médicale

Indiana Biosciences Research Institute

Welbio/FRFS

Agence Nationale de la Recherche

EU IMI2

Publisher

Springer Science and Business Media LLC

Subject

Endocrinology, Diabetes and Metabolism,Internal Medicine

Link

https://link.springer.com/content/pdf/10.1007/s00125-020-05176-7.pdf

Reference62 articles.

1. Campbell-Thompson M, Fu A, Kaddis JS et al (2016) Insulitis and beta-cell mass in the natural history of type 1 diabetes. Diabetes 65(3):719–731. https://doi.org/10.2337/db15-0779

2. Santin I, Eizirik DL (2013) Candidate genes for type 1 diabetes modulate pancreatic islet inflammation and beta-cell apoptosis. Diabetes Obes Metab 15(Suppl 3):71–81. https://doi.org/10.1111/dom.12162

3. Ramos-Rodriguez M, Raurell-Vila H, Colli ML et al (2019) The impact of proinflammatory cytokines on the beta-cell regulatory landscape provides insights into the genetics of type 1 diabetes. Nat Genet 51(11):1588–1595. https://doi.org/10.1038/s41588-019-0524-6

4. Marroqui L, Santin I, Dos Santos RS, Marselli L, Marchetti P, Eizirik DL (2014) BACH2, a candidate risk gene for type 1 diabetes, regulates apoptosis in pancreatic beta-cells via JNK1 modulation and crosstalk with the candidate gene PTPN2. Diabetes 63(7):2516–2527. https://doi.org/10.2337/db13-1443

5. Moore F, Colli ML, Cnop M et al (2009) PTPN2, a candidate gene for type 1 diabetes, modulates interferon-gamma-induced pancreatic beta-cell apoptosis. Diabetes 58(6):1283–1291. https://doi.org/10.2337/db08-1510

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