HPV18 Utilizes Two Alternative Branch Sites for E6*I Splicing to Produce E7 Protein
Author:
Publisher
Elsevier BV
Subject
Virology,Molecular Medicine,Immunology
Link
http://link.springer.com/content/pdf/10.1007/s12250-019-00098-0.pdf
Reference42 articles.
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2. Ajiro M, Zheng ZM (2014) Oncogenes and RNA splicing of human tumor viruses. Emerg Microbes Infect 3:e63
3. Ajiro M, Zheng ZM (2015a) E6^E7, a novel splice isoform protein of human papillomavirus 16, stabilizes viral E6 and E7 oncoproteins via HSP90 and GRP78. mBio 6:e02014–e02068
4. Ajiro M, Zheng ZM (2015b) Vemurafenib-resistant BRAF selects alternative branch points different from its wild-type BRAF in intron 8 for RNA splicing. Cell Biosci 5:70
5. Ajiro M, Jia R, Zhang L, Liu X, Zheng ZM (2012) Intron definition and a branch site adenosine at nt 385 control RNA splicing of HPV16 E6*I and E7 expression. PLoS ONE 7:e46412
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