The Role of Alternative Splicing in Cancer: Regulatory Mechanism, Therapeutic Strategy, and Bioinformatics Application
Author:
Affiliation:
1. Department of Pathology, Laboratory of Clinical and Experimental Pathology, Xuzhou Medical University, Xuzhou, China.
2. Department of General Surgery, Xuzhou Medical University, Xuzhou, China.
Publisher
Mary Ann Liebert Inc
Subject
Cell Biology,Genetics,Molecular Biology,General Medicine
Link
https://www.liebertpub.com/doi/pdf/10.1089/dna.2022.0322
Reference301 articles.
1. A VEGF-A/SOX2/SRSF2 network controls VEGFR1 pre-mRNA alternative splicing in lung carcinoma cells
2. Meayamycin inhibits pre–messenger RNA splicing and exhibits picomolar activity against multidrug-resistant cells
3. SRSF1-Regulated Alternative Splicing in Breast Cancer
4. A hypoxia-inducible factor (HIF)-3α splicing variant, HIF-3α4 impairs angiogenesis in hypervascular malignant meningiomas with epigenetically silenced HIF-3α4
5. N-terminal phosphorylation of glutaminase C decreases its enzymatic activity and cancer cell migration
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