Stress Granule Formation is One of the Early Antiviral Mechanisms for Host Cells Against Coxsackievirus B Infection
Author:
Publisher
Elsevier BV
Subject
Virology,Molecular Medicine,Immunology
Link
http://link.springer.com/article/10.1007/s12250-018-0040-3/fulltext.html
Reference34 articles.
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3. Chau DH, Yuan J, Zhang H, Cheung P, Lim T, Liu Z, Sall A, Yang D (2007) Coxsackievirus B3 proteases 2A and 3C induce apoptotic cell death through mitochondrial injury and cleavage of eIF4GI but not DAP5/p97/NAT1. Apoptosis 12:513–524
4. Choudhury P, Bussiere L, Miller CL (2017) Mammalian orthoreovirus factories modulate stress granule protein localization by interaction with G3BP1. J Virol 91:e01298–e01317
5. Dougherty JD, White JP, Lloyd RE (2011) Poliovirus-mediated disruption of cytoplasmic processing bodies. J Virol 85:64–75
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1. Pro-Viral and Anti-Viral Roles of the RNA-Binding Protein G3BP1;Viruses;2023-02-06
2. Stress Granules Inhibit Coxsackievirus B3-Mediated Cell Death via Reduction of Mitochondrial Reactive Oxygen Species and Viral Extracellular Release;Journal of Microbiology and Biotechnology;2023-01-11
3. The PERK/PKR-eIF2α Pathway Negatively Regulates Porcine Hemagglutinating Encephalomyelitis Virus Replication by Attenuating Global Protein Translation and Facilitating Stress Granule Formation;Journal of Virology;2022-01-12
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