Termination of Transcription of LAT Increases the Amounts of ICP0 mRNA but Does Not Alter the Course of HSV-1 Infection in Latently Infected Murine Ganglia-Reference-Cited by-同舟云学术

Termination of Transcription of LAT Increases the Amounts of ICP0 mRNA but Does Not Alter the Course of HSV-1 Infection in Latently Infected Murine Ganglia

Published:2020-09-07 Issue:2 Volume:36 Page:264-272
ISSN:1674-0769
Container-title:Virologica Sinica
language:en
Short-container-title:Virol. Sin.

Author:

Jiang Haifang,Wu Jiaming,Liu Xianjie,Lu Ruitao,Zhou Manling,Chen Meiling,Liu Yonghong,Zhou Grace Guoying^ORCID,Fu Wenmin^ORCID

Publisher

Elsevier BV

Subject

Virology,Molecular Medicine,Immunology

Link

https://link.springer.com/content/pdf/10.1007/s12250-020-00287-2.pdf

Reference28 articles.

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3. Chen SH, Lee LY, Garber DA, Schaffer PA, Knipe DM, Coen DM (2002) Neither LAT nor open reading frame P mutations increase expression of spliced or intron-containing ICP0 transcripts in mouse ganglia latently infected with herpes simplex virus. J Virol 76:4764–4772

4. Devi-Rao GB, Goodart SA, Hecht LM, Rochford R, Rice MK, Wagner EK (1991) Relationship between polyadenylated and nonpolyadenylated herpes simplex virus type 1 latency-associated transcripts. J Virol 65:2179–2190

5. Du T, Han Z, Zhou G, Roizman B (2015) Patterns of accumulation of miRNAs encoded by herpes simplex virus during productive infection, latency, and on reactivation. Proc Natl Acad Sci USA 112:E49–55