Establishment of a cell model of ALS disease: Golgi apparatus disruption occurs independently from apoptosis-Reference-Cited by-同舟云学术

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Establishment of a cell model of ALS disease: Golgi apparatus disruption occurs independently from apoptosis

Published:2007-11-15 Issue:4 Volume:30 Page:603-610
ISSN:0141-5492
Container-title:Biotechnology Letters
language:en
Short-container-title:Biotechnol Lett

Author:

Gomes Catarina,Palma Angelina S.,Almeida Rui,Regalla Manuela,McCluskey Leo F.,Trojanowski John Q.,Costa Júlia

Publisher

Springer Science and Business Media LLC

Subject

General Medicine,Biotechnology,Bioengineering,Applied Microbiology and Biotechnology

Link

http://link.springer.com/content/pdf/10.1007/s10529-007-9595-z.pdf

Reference20 articles.

1. Brito C, Escrevente C, Reis CA et al (2007) Increased levels of fucosyltransferase IX and carbohydrate Lewis(x) adhesion determinant in human NT2N neurons. J Neurosci Res 85:1260–1270

2. Bruijn LI, Miller TM, Cleveland DW (2004) Unraveling the mechanisms involved in motor neuron degeneration in ALS. Annu Rev Neurosci 27:723–749

3. Cashman NR, Durham HD, Blusztajn JK et al (1992) Neuroblastoma × spinal cord (NSC) hybrid cell lines resemble developing motor neurons. Dev Dyn 194:209–221

4. Cooper AA, Gitler AD, Cashikar A et al (2006) Alpha-synuclein blocks ER-Golgi traffic and Rab1 rescues neuron loss in Parkinson’s models. Science 313:324–328

5. Demestre M, Parkin-Smith G, Petzold A et al (2005) The pro and the active form of matrix metalloproteinase-9 is increased in serum of patients with amyotrophic lateral sclerosis. J Neuroimmunol 159:146–154

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