Lack of β-amyloid cleaving enzyme-1 (BACE1) impairs long-term synaptic plasticity but enhances granule cell excitability and oscillatory activity in the dentate gyrus in vivo
Author:
Funder
Deutsche Forschungsgemeinschaft
Alzheimer Forschung Initiative
Publisher
Springer Science and Business Media LLC
Subject
Histology,General Neuroscience,Anatomy
Link
http://link.springer.com/article/10.1007/s00429-019-01836-6/fulltext.html
Reference86 articles.
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2. Blume T, Filser S, Jaworska A et al (2018) BACE1 Inhibitor MK-8931 alters formation but not stability of dendritic spines. Front Aging Neurosci 10:229. https://doi.org/10.3389/fnagi.2018.00229
3. Bowden JB, Abraham WC, Harris KM (2012) Differential effects of strain, circadian cycle, and stimulation pattern on LTP and concurrent LTD in the dentate gyrus of freely moving rats. Hippocampus 22:1363–1370. https://doi.org/10.1002/hipo.20972
4. Bronzino JD, Abu-Hasaballah K, Austin-LaFrance RJ, Morgane PJ (1994) Maturation of long-term potentiation in the hippocampal dentate gyrus of the freely moving rat. Hippocampus 4:439–446. https://doi.org/10.1002/hipo.450040406
5. Bronzino JD, Blaise JH, Morgane PJ (1997) The paired-pulse index: a measure of hippocampal dentate granule cell modulation. Ann Biomed Eng 25:870–873
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