EGCG attenuate EGF triggered matrix abundance and migration in HPV positive and HPV negative cervical cancer cells
Author:
Funder
KAHE DST
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Oncology,Hematology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s12032-023-02135-1.pdf
Reference34 articles.
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2. Hugo de Almeida V, Guimarães IDS, Almendra LR, Rondon AMR, Tilli TM, de Melo AC, Sternberg C, Monteiro RQ. Positive crosstalk between EGFR and the TF-PAR2 pathway mediates resistance to cisplatin and poor survival in cervical cancer. Oncotarget. 2018;9(55):30594–609. https://doi.org/10.18632/oncotarget.25748.
3. Muthusami S, Prabakaran DS, Yu JR, Park WY. EGF-induced expression of Fused Toes Homolog (FTS) facilitates epithelial–mesenchymal transition and promotes cell migration in ME180 cervical cancer cells. Cancer Lett. 2014;351(2):252–9. https://doi.org/10.1016/j.canlet.2014.06.007.
4. Thomas R, Weihua Z. Rethink of EGFR in cancer with its kinase independent function on board. Front Oncol. 2019;9:800. https://doi.org/10.3389/fonc.2019.00800.
5. Cai WQ, Zeng LS, Wang LF, Wang YY, Cheng JT, Zhang Y, Han ZW, Zhou Y, Huang SL, Wang XW, Peng XC, Xiang Y, Ma Z, Cui SZ, Xin HW. The latest battles between EGFR monoclonal antibodies and resistant tumor cells. Front Oncol. 2020;10:1249. https://doi.org/10.3389/fonc.2020.01249.
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