Caveolar disruption with methyl-β-cyclodextrin causes endothelium-dependent contractions in Wistar rat carotid arteries
Author:
Publisher
Springer Science and Business Media LLC
Subject
Health, Toxicology and Mutagenesis,Pollution,Environmental Chemistry,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s11356-022-20226-w.pdf
Reference53 articles.
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2. Ahmad A, Dempsey SK, Daneva Z, Azam M, Li N, Li P-L, Ritter JK (2018) Role of nitric oxide in the cardiovascular and renal systems. Int J Mol Sci 19:2605
3. Al-Brakati A, Kamishima T, Dart C, Quayle J (2015) Caveolar disruption causes contraction of rat femoral arteries via reduced basal NO release and subsequent closure of BKCa channels. PeerJ 3:e966
4. Andjelkovic AV, Stamatovic SM, Phillips CM, Martinez-Revollar G, Keep RF (2020) Modeling blood–brain barrier pathology in cerebrovascular disease in vitro: current and future paradigms. Fluids and Barriers of the CNS 17:1–21
5. Ando M, Matsumoto T, Kobayashi S, Iguchi M, Taguchi K, Kobayashi T (2018) Differential participation of calcium-activated potassium channel in endothelium-dependent hyperpolarization-type relaxation in superior mesenteric arteries of spontaneously hypertensive rats. Can J Physiol Pharmacol 96:839–844
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