Ouabain Regulates CFTR-Mediated Anion Secretion and Na,K-ATPase Transport in ADPKD Cells-Reference-Cited by-同舟云学术

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Ouabain Regulates CFTR-Mediated Anion Secretion and Na,K-ATPase Transport in ADPKD Cells

Published:2015-08-20 Issue:6 Volume:248 Page:1145-1157
ISSN:0022-2631
Container-title:The Journal of Membrane Biology
language:en
Short-container-title:J Membrane Biol

Author:

Jansson Kyle,Venugopal Jessica,Sánchez Gladis,Magenheimer Brenda S.,Reif Gail A.,Wallace Darren P.,Calvet James P.,Blanco Gustavo

Funder

National Institutes of Health

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Physiology,Biophysics

Link

http://link.springer.com/content/pdf/10.1007/s00232-015-9832-7.pdf

Reference53 articles.

1. Bagrov AY, Shapiro JI, Fedorova OV (2009) Endogenous cardiotonic steroids: physiology, pharmacology, and novel therapeutic targets. Pharmacol Rev 61:9–38. doi: 10.1124/pr.108.000711

2. Belibi FA et al (2004) Cyclic AMP promotes growth and secretion in human polycystic kidney epithelial cells. Kidney Int 66:964–973. doi: 10.1111/j.1523-1755.2004.00843.x

3. Blanco G, Mercer RW (1998) Isozymes of the Na,K-ATPase: heterogeneity in structure. Am J Physiol 275:F633–F650

4. Blanco G, Wallace DP (2013) Novel role of ouabain as a cystogenic factor in autosomal dominant polycystic kidney disease. Am J Physiol Renal Physiol 305:F797–F812. doi: 10.1152/ajprenal.00248.2013

5. Blaustein MP, Hamlyn JM (2010) Signaling mechanisms that link salt retention to hypertension: endogenous ouabain, the Na(+) pump, the Na(+)/Ca(2+) exchanger and TRPC proteins. Biochim Biophys Acta 1802:1219–1229. doi: 10.1016/j.bbadis.2010.02.011

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2. Ouabain enhances renal cyst growth in a slowly progressive mouse model of autosomal dominant polycystic kidney disease;American Journal of Physiology-Renal Physiology;2023-12-01

3. Ouabain-Induced Gene Expression Changes in Human iPSC-Derived Neuron Culture Expressing Dopamine and cAMP-Regulated Phosphoprotein 32 and GABA Receptors;Brain Sciences;2021-02-07

4. Targeting chloride transport in autosomal dominant polycystic kidney disease;Cellular Signalling;2020-09

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