Lipid Peroxidation, Protein Thiols and Calcium Homeostasis in Bromobenzene-Induced Liver Damage

Author:

Casini A. F.,Maellaro E.,Pompella A.,Ferrali M.,Comporti M.

Publisher

Springer US

Reference10 articles.

1. A. F. Casini, A. Pompella, and M. Comporti, Liver glutathione induced by bromobenzene, iodobenzene, and diethylmaleate poisoning and its relation to lipid peroxidation and necrosis, Am.J.Path. 118:225 (1985).

2. G. Bellomo, S. A. Jewell, M. T. Smith, H. Thor, and S. Orrenius, Perturbation of Ca2+ homeostasis during hepatocyte injury, in: “Mechanisms of Hepatocyte Injury and Death,” D. Keppler, H. Popper, L. Bianchi, and W. Reutter, eds., MTP Press Limited, Lancaster (1984).

3. G. Bellomo and S. Orrenius, Altered thiol and calcium homeostasis in oxidative hepatocellular injury, Hepatology 5:876 (1985).

4. D. Di Monte, G. Bellomo, H. Thor, P. Nicotera, and S. Orrenius, Menadione-induced cytotoxicity is aassociated with protein thiol oxidation and alteration in intracellular Ca2+ homeostasis, Archs.Biochem.Biophys. 235:343 (1984).

5. M. Moore, H. Thor, G. Moore, S. Nelson, P. Moldeus, and S. Orrenius, The toxicity of acetaminophen and N-acetyl-p-benzoquinone imine in isolated hepatocytes is associated with thiol depletion and increased cytosolic Ca2+, J.Biol.Chem. 260:13035 (1985).

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