Antibiotic anisomycin induces cell cycle arrest and apoptosis through inhibiting mitochondrial biogenesis in osteosarcoma
Author:
Funder
Xiangyang Central Hospital
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Physiology
Link
http://link.springer.com/article/10.1007/s10863-017-9734-8/fulltext.html
Reference24 articles.
1. Abarrategi A, Tornin J, Martinez-Cruzado L, Hamilton A, Martinez-Campos E, Rodrigo JP et al (2016) Osteosarcoma: cells-of-origin, cancer stem cells, and targeted therapies. Stem Cells Int 2016:3631764. https://doi.org/10.1155/2016/3631764.
2. Abayasiriwardana KS, Barbone D, Kim KU, Vivo C, Lee KK, Dansen TB et al (2007) Malignant mesothelioma cells are rapidly sensitized to TRAIL-induced apoptosis by low-dose anisomycin via Bim. Mol Cancer Ther 6(10):2766–2776. https://doi.org/10.1158/1535-7163.MCT-07-0278
3. Angulo P, Kaushik G, Subramaniam D, Dandawate P, Neville K, Chastain K et al (2017) Natural compounds targeting major cell signaling pathways: a novel paradigm for osteosarcoma therapy. J Hematol Oncol 10(1):10. https://doi.org/10.1186/s13045-016-0373-z.
4. Barros LF, Young M, Saklatvala J, Baldwin SA (1997) Evidence of two mechanisms for the activation of the glucose transporter GLUT1 by anisomycin: p38(MAP kinase) activation and protein synthesis inhibition in mammalian cells. J Physiol 504(Pt 3):517–525
5. Bhat M, Robichaud N, Hulea L, Sonenberg N, Pelletier J, Topisirovic I (2015) Targeting the translation machinery in cancer. Nat Rev Drug Discov 14(4):261–278. https://doi.org/10.1038/nrd4505
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