Evidence of two mechanisms for the activation of the glucose transporter GLUT1 by anisomycin: p38(MAP kinase) activation and protein synthesis inhibition in mammalian cells
Author:
Publisher
Wiley
Subject
Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1469-7793.1997.517bd.x/fullpdf
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3. Anisomycin-activated protein kinases p45 and p55 but not mitogen-activated protein kinases ERK-1 and −2 are implicated in the induction of c-fos and c-jun;Cano;Molecular and Cellular Biology,1994
4. Parallel signal processing among mammalian MAPKs;Cano;Trends in Biochemical Sciences,1995
5. Protein synthesis inhibitors activate glucose transport without increasing plasma membrane glucose transporters in 3T3-L1 adipocytes;Clancy;Journal of Biological Chemistry,1991
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