The BM2 protein of influenza B virus interacts with p53 and inhibits its transcriptional and apoptotic activities-Reference-Cited by-同舟云学术

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The BM2 protein of influenza B virus interacts with p53 and inhibits its transcriptional and apoptotic activities

Published:2015-02-11 Issue:1-2 Volume:403 Page:187-197
ISSN:0300-8177
Container-title:Molecular and Cellular Biochemistry
language:en
Short-container-title:Mol Cell Biochem

Author:

Zhang H.,Yu H.,Wang J.,Zhang M.,Wang X.,Ahmad W.,Duan M.,Guan Z.

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,General Medicine

Link

http://link.springer.com/content/pdf/10.1007/s11010-015-2349-7.pdf

Reference43 articles.

1. Takeda M, Pekosz A, Shuck K, Pinto LH, Lamb RA (2002) Influenza A virus M2 ion channel activity is essential for efficient replication in tissue culture. J Virol 76:1391–1399. doi: 10.1128/jvi.76.3.1391-1399.2002

2. Jackson D, Cadman A, Zurcher T, Barclay WS (2002) A reverse genetics approach for recovery of recombinant influenza B viruses entirely from cDNA. J Virol 76:11744–11747. doi: 10.1128/jvi.76.22.11744-11747.2002

3. Briedis DJ, Lamb RA, Choppin PW (1982) Sequence of RNA segment 7 of the influenza B virus genome: partial amino acid homology between the membrane proteins (M1) of influenza A and B viruses and conservation of a second open reading frame. Virology 116:581–588. doi: 10.1016/0042-6822(82)90150-7

4. Pinto LH, Lamb RA (2006) The M2 proton channels of influenza A and B viruses. J Biol Chem 281:8997–9000. doi: 10.1074/jbc.R500020200

5. Odagiri T, Hong J, Ohara Y (1999) The BM2 protein of influenza B virus is synthesized in the late phase of infection and incorporated into virions as a subviral component. J Gen Virol 80:2573–2581

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