Chrysin protects cardiac H9c2 cells against H2O2-induced endoplasmic reticulum stress by up-regulating the Nrf2/PERK pathway-Reference-Cited by-同舟云学术

Chrysin protects cardiac H9c2 cells against H2O2-induced endoplasmic reticulum stress by up-regulating the Nrf2/PERK pathway

Published:2022-08-09 Issue:3 Volume:478 Page:539-553
ISSN:0300-8177
Container-title:Molecular and Cellular Biochemistry
language:en
Short-container-title:Mol Cell Biochem

Author:

Yuvaraj Subramani,Ajeeth Arumugam Kalaiselvi,Puhari Shanavas Syed Mohamed,Abhishek Albert,Ramprasath Tharmarajan,Vasudevan Varadaraj,Vignesh Narasimman,Selvam Govindan Sadasivam^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,General Medicine

Link

https://link.springer.com/content/pdf/10.1007/s11010-022-04531-z.pdf

Reference47 articles.

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3. Bundhun PK, Wu ZJ, Chen MH (2015) Impact of modifiable cardiovascular risk factors on mortality after percutaneous coronary intervention: a systematic review and meta-analysis of 100 studies. Medicine (Baltimore) 94:e2313. https://doi.org/10.1097/MD.0000000000002313

4. Horke S, Witte I, Wilgenbus P, Kruger M, Strand D, Forstermann U (2007) Paraoxonase-2 reduces oxidative stress in vascular cells and decreases endoplasmic reticulum stress-induced caspase activation. Circulation 115:2055–2064. https://doi.org/10.1161/CIRCULATIONAHA.106.681700

5. Park WJ, Park JW (2020) The role of sphingolipids in endoplasmic reticulum stress. FEBS Lett 594:3632–3651. https://doi.org/10.1002/1873-3468.13863

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