Modulation of p53, c-fos, RARE, cyclin A, and cyclin D1 expression in human leukemia (HL-60) cells exposed to arsenic trioxide
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Clinical Biochemistry,Molecular Biology,General Medicine
Link
http://link.springer.com/content/pdf/10.1007/s11010-009-0160-z.pdf
Reference57 articles.
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2. Rego EM, He LZ, Warrell RP Jr, Wang ZG, Pandolfi PP (2000) Retinoic acid (RA) and As2O3 treatment in transgenic models of acute promyelocytic leukemia (APL) unravel the distinct nature of the leukemiamogenic process induced by the PML-RARapha and PLZF-RARalpha oncoproteins. Proc Natl Acad Sci USA 97:10173–10178. doi: 10.1073/pnas.180290497
3. Zhu J, Koken MHM, Quignon F, Chelbi-Alix MK, Degos L, Wang ZY, Chen Z, de Thé H (1997) Arsenic-induced PML targeting onto nuclear bodies: implications for the treatment of acute promyelocytic leukemia. Proc Natl Acad Sci USA 94:3978–3983. doi: 10.1073/pnas.94.8.3978
4. Bode A, Dong Z (2000) Apoptosis induction by arsenic: mechanisms of action and possible clinical applications for treating therapy-resistant cancers. J Drug Resist Updat 3:21–29. doi: 10.1054/drup.2000.0114
5. Bode AM, Dong Z (2002) The paradox of arsenic: molecular mechanisms of cell transformation and chemotherapeutic effects. Crit Rev Oncol Hematol 42:5–24. doi: 10.1016/S1040-8428(01)00215-3
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