Possible role of mitochondrial K-ATP channel and nitric oxide in protection of the neonatal rat heart
Author:
Funder
Grantová Agentura České Republiky
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Clinical Biochemistry,Molecular Biology,General Medicine
Link
http://link.springer.com/article/10.1007/s11010-018-3370-4/fulltext.html
Reference39 articles.
1. Ostadal B, Ostadalova I, Dhalla NS (1999) Development of cardiac sensitivity to oxygen deficiency: comparative and ontogenetic aspects. Physiol Rev 79(3):635–659
2. Ostadal B, Netuka I, Maly J, Besik J, Ostadalova I (2009) Gender differences in cardiac ischemic injury and protection–experimental aspects. Exp Biol Med (Maywood) 234(9):1011–1019. https://doi.org/10.3181/0812-MR-362
3. Ostadalova I, Ostadal B, Kolar F, Parratt JR, Wilson S (1998) Tolerance to ischaemia and ischaemic preconditioning in neonatal rat heart. J Mol Cell Cardiol 30(4):857–865. https://doi.org/10.1006/jmcc.1998.0653
4. Ostadalova I, Ostadal B, Jarkovska D, Kolar F (2002) Ischemic preconditioning in chronically hypoxic neonatal rat heart. Pediatr Res 52(4):561–567. https://doi.org/10.1203/00006450-200210000-00016
5. Doul J, Charvatova Z, Ostadalova I, Kohutiar M, Maxova H, Ostadal B (2015) Neonatal rat hearts cannot be protected by ischemic postconditioning. Physiol Res 64(6):789–794
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