Myosin heavy chain and cardiac troponin T damage is associated with impaired myofibrillar ATPase activity contributing to sarcomeric dysfunction in Ca2+-paradox rat hearts-Reference-Cited by-同舟云学术

Myosin heavy chain and cardiac troponin T damage is associated with impaired myofibrillar ATPase activity contributing to sarcomeric dysfunction in Ca2+-paradox rat hearts

Published:2017-02-17 Issue:1-2 Volume:430 Page:57-68
ISSN:0300-8177
Container-title:Molecular and Cellular Biochemistry
language:en
Short-container-title:Mol Cell Biochem

Author:

Kovács Árpád,Kalász Judit,Pásztor Enikő T.,Tóth Attila,Papp Zoltán,Dhalla Naranjan S.,Barta Judit^ORCID

Funder

Canadian Institutes of Health Research

Országos Tudományos Kutatási Alapprogramok

New National Excellence Program of the Ministry of Human Capacities

TACTICS program, in association with CIHR and Heart and Stroke Foundation of Canada

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,General Medicine

Link

http://link.springer.com/article/10.1007/s11010-017-2954-8/fulltext.html

Reference39 articles.

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2. Zimmerman AN, Daems W, Hülsmann WC, Snijder J, Wisse E, Durrer D (1967) Morphological changes of heart muscle caused by successive perfusion with calcium-free and calcium-containing solutions (calcium paradox). Cardiovasc Res 1(3):201–209

3. Steenbergen C, Murphy E, Watts JA, London RE (1990) Correlation between cytosolic free calcium, contracture, ATP, and irreversible ischemic injury in perfused rat heart. Circ Res 66(1):135–146

4. Piper HM (2000) The calcium paradox revisited: an artefact of great heuristic value. Cardiovasc Res 45(1):123–127

5. Zhang M, Xu YJ, Saini HK, Turan B, Liu PP, Dhalla NS (2005) TNF-alpha as a potential mediator of cardiac dysfunction due to intracellular Ca2+-overload. Biochem Biophys Res Commun 327(1):57–63

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