Proteasome Inhibitors Prevent the Degradation of Familial Alzheimer’s Disease-Linked Presenilin 1 and Potentiate Aβ42 Recovery from Human Cells-Reference-Cited by-同舟云学术

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Proteasome Inhibitors Prevent the Degradation of Familial Alzheimer’s Disease-Linked Presenilin 1 and Potentiate Aβ42 Recovery from Human Cells

Published:1998-03 Issue:3 Volume:4 Page:147-157
ISSN:1076-1551
Container-title:Molecular Medicine
language:en
Short-container-title:Mol Med

Author:

Marambaud Philippe,Ancolio Karine,Lopez-Perez Elvira,Checler Frédéric

Publisher

Springer Science and Business Media LLC

Subject

Genetics (clinical),Genetics,Molecular Biology,Molecular Medicine

Link

http://link.springer.com/content/pdf/10.1007/BF03401912.pdf

Reference40 articles.

1. Lemere CA, Lopera F, Kosik KS, et al. (1996) The E280A presenilin 1 Alzheimer mutation produces increased Aβ42 deposition and severe cerebellar pathology. Nat. Med. 2: 1146–1150.

2. Mann DMA, Iwatsubo T, Cairns NJ, et al. (1997) Amyloid β protein (Aβ) deposition in chromosome 14-linked Alzheimer’s disease: Predominance of Aβ42(43). Ann. Neurol. 40: 149–156.

3. Mann DMA, Iwatsubo T, Nochlin D, Sumi SM, Levy-Lahad E, Bird TD. (1997) Amyloid (Aβ) deposition in Chromosome 1-linked Alzheimer’s disease: The Volga German family. Ann. Neurol. 41: 52–57.

4. Borchelt DR, Thinakaran G, Eckman CB, et al. (1996) Familial Alzheimer’s disease-linked presenilin 1 variants elevate Aβ1-42/1-40 in vitro and in vivo. Neuron 17: 1005–1013.

5. Gearing M, Tigges J, Mori H, Mirra SS. (1996) Aβ40 is a major form of β-amyloid in human primates. Neurobiol. Aging 17: 903–908.

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