H3.3 K27M depletion increases differentiation and extends latency of diffuse intrinsic pontine glioma growth in vivo-Reference-Cited by-同舟云学术

H3.3 K27M depletion increases differentiation and extends latency of diffuse intrinsic pontine glioma growth in vivo

Published:2019-02-15 Issue:4 Volume:137 Page:637-655
ISSN:0001-6322
Container-title:Acta Neuropathologica
language:en
Short-container-title:Acta Neuropathol

Author:

Silveira André B.,Kasper Lawryn H.,Fan Yiping,Jin Hongjian,Wu Gang,Shaw Timothy I.,Zhu Xiaoyan,Larson Jon D.,Easton John,Shao Ying,Yergeau Donald A.,Rosencrance Celeste,Boggs Kristy,Rusch Michael C.,Ding Liang,Zhang Junyuan,Finkelstein David,Noyes Rachel M.,Russell Brent L.,Xu Beisi,Broniscer Alberto,Wetmore Cynthia,Pounds Stanley B.,Ellison David W.,Zhang Jinghui,Baker Suzanne J.

Funder

National Institutes of Health

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Neurology (clinical),Pathology and Forensic Medicine

Link

http://link.springer.com/article/10.1007/s00401-019-01975-4/fulltext.html

Reference55 articles.

1. Ashburner M, Ball CA, Blake JA, Botstein D, Butler H, Cherry JM et al (2000) Gene ontology: tool for the unification of biology. The gene ontology consortium. Nat Genet 25:25–29. https://doi.org/10.1038/75556

2. Barbie DA, Tamayo P, Boehm JS, Kim SY, Moody SE, Dunn IF et al (2009) Systematic RNA interference reveals that oncogenic KRAS-driven cancers require TBK1. Nature 462:108–112. https://doi.org/10.1038/nature08460

3. Beguelin W, Popovic R, Teater M, Jiang Y, Bunting KL, Rosen M et al (2013) EZH2 is required for germinal center formation and somatic EZH2 mutations promote lymphoid transformation. Cancer Cell 23:677–692. https://doi.org/10.1016/j.ccr.2013.04.011