Casein kinase-1 isoforms differentially associate with neurofibrillary and granulovacuolar degeneration lesions-Reference-Cited by-同舟云学术

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Casein kinase-1 isoforms differentially associate with neurofibrillary and granulovacuolar degeneration lesions

Published:2006-03-24 Issue:5 Volume:111 Page:413-421
ISSN:0001-6322
Container-title:Acta Neuropathologica
language:en
Short-container-title:Acta Neuropathol

Author:

Kannanayakal Theresa J.,Tao Haiyang,Vandre Dale D.,Kuret Jeff

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Neurology (clinical),Pathology and Forensic Medicine

Link

http://link.springer.com/content/pdf/10.1007/s00401-006-0049-9.pdf

Reference62 articles.

1. Abdel-Sater F, El Bakkoury M, Urrestarazu A, Vissers S, Andre B (2004) Amino acid signaling in yeast: casein kinase I and the Ssy5 endoprotease are key determinants of endoproteolytic activation of the membrane-bound Stp1 transcription factor. Mol Cell Biol 24:9771–9785

2. Amit S, Hatzubai A, Birman Y, Andersen JS, Ben-Shushan E, Mann M, Ben-Neriah Y, Alkalay I (2002) Axin-mediated CKI phosphorylation of b-catenin at Ser 45: a molecular switch for the Wnt pathway. Genes Dev 16:1066–1076

3. Ball MJ (1977) Neuronal loss, neurofibrillary tangles and granulovacuolar degeneration in the hippocampus with ageing and dementia. A quantitative study. Acta Neuropathol (Berl) 37:111–118

4. Ball MJ (1978) Topographic distribution of neurofibrillary tangles and granulovacuolar degeneration in hippocampal cortex of aging and demented patients. A quantitative study. Acta Neuropathol (Berl) 42:73–80

5. Ball MJ, Nuttall K (1981) Topography of neurofibrillary tangles and granulovacuoles in hippocampi of patients with Down’s syndrome: quantitative comparison with normal ageing and Alzheimer’s disease. Neuropathol Appl Neurobiol 7:13–20

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