The Alzheimer’s β-secretase BACE1 localizes to normal presynaptic terminals and to dystrophic presynaptic terminals surrounding amyloid plaques-Reference-Cited by-同舟云学术

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The Alzheimer’s β-secretase BACE1 localizes to normal presynaptic terminals and to dystrophic presynaptic terminals surrounding amyloid plaques

Published:2013-07-03 Issue:3 Volume:126 Page:329-352
ISSN:0001-6322
Container-title:Acta Neuropathologica
language:en
Short-container-title:Acta Neuropathol

Author:

Kandalepas Patty C.,Sadleir Katherine R.,Eimer William A.,Zhao Jie,Nicholson Daniel A.,Vassar Robert

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Clinical Neurology,Pathology and Forensic Medicine

Link

http://link.springer.com/article/10.1007/s00401-013-1152-3/fulltext.html

Reference123 articles.

1. Adalbert R, Nogradi A, Babetto E, Janeckova L, Walker SA, Kerschensteiner M, Misgeld T, Coleman MP (2009) Severely dystrophic axons at amyloid plaques remain continuous and connected to viable cell bodies. Brain J Neurol 132(Pt 2):402–416

2. Amaral DG, Dent JA (1981) Development of the mossy fibers of the dentate gyrus: I. A light and electron microscopic study of the mossy fibers and their expansions. J Comp Neurol 195(1):51–86

3. Barth S, Glick D, Macleod KF (2010) Autophagy: assays and artifacts. J Pathol 221(2):117–124

4. Bednarski E, Ribak CE, Lynch G (1997) Suppression of cathepsins B and L causes a proliferation of lysosomes and the formation of mega neurites in hippocampus. J Neurosci 17(11):4006–4021

5. Bi X, Zhou J, Lynch G (1999) Lysosomal protease inhibitors induce mega neurites and tangle-like structures in entorhinohippocampal regions vulnerable to Alzheimer’s disease. Exp Neurol 158(2):312–327

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