The salt paradox and its possible implications in managing hypertensive diabetic patients
Author:
Publisher
Springer Science and Business Media LLC
Subject
Internal Medicine
Link
http://link.springer.com/content/pdf/10.1007/s11906-005-0089-x.pdf
Reference49 articles.
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2. Abbate M, Zoja C, Corna D, et al.: In progressive nephropathies, overload of tubular cells with filtered proteins translates glomerular permeability dysfunction into cellular signals of interstitial inflammation. J Am Soc Nephrol 1998, 9:1213–1224.
3. Abbate M, Remuzzi G: Proteinuria as a mediator of tubulointerstitial injury. Kidney Blood Press Res 1999, 22:37–46.
4. Mauer SM, Steffes MW, Ellis EN, et al.: Structural functional relationships in diabetic nephropathy. J Clin Invest 1984, 74:1143–1155.
5. Bohle A, Mackensen Haen S, von Gise H, et al.: The consequences of tubulointerstitial changes for renal function in glomerulopathies: a morphometric and cytological analysis. Pathol Res Pract 1990, 186:135–144.
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