A new calpain inhibitor protects left ventricular dysfunction induced by mild ischemia-reperfusion in in situ rat hearts
Author:
Publisher
Springer Science and Business Media LLC
Subject
Physiology
Link
http://link.springer.com/content/pdf/10.1007/s12576-012-0243-6.pdf
Reference31 articles.
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2. Karmazyn M, Gan XT, Humphreys RA, Yoshida H, Kusumoto K (1999) The myocardial Na+/H+ exchange structure, regulation, and its role in heart disease. Circ Res 85:777–786
3. Schäfer C, Ladilov Y, Inserte J, Schäfer M, Haffner S, Garcia-Dorado D, Piper HM (2001) Role of the reverse mode of the Na+/Ca2+ exchanger in reoxygenation-induced cardiomyocyte injury. Cardiovasc Res 51:241–250
4. Tani M, Neely JR (1989) Role of intracellular Na+ in Ca2+ overload and depressed recovery of ventricular function of reperfused ischemic rat hearts. Possible involvement of H+–Na+ and Na+–Ca2+ exchange. Circ Res 65:1045–1056
5. Kobayashi S, Yoshikawa Y, Sakata S, Takenaka C, Hagihara H, Ohga Y, Abe T, Taniguchi S, Takaki M (2004) Left ventricular mechanoenergetics after hyperpolarized cardioplegic arrest by nicorandil and after depolarized cardioplegic arrest by KCl. Am J Physiol Heart Circ Physiol 287:H1072–H1080
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