Enhanced sympathetic activity in young spontaneously hypertensive rats is not the trigger mechanism for genetic hypertension

Author:

Nakamura Keiji,Nakamura Kazuo

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology,General Medicine

Reference38 articles.

1. Anton, A. H., Sayre, D. F.: A study of the factors affecting the aluminium oxide-trihydroxyindole procedure for the analysis of catecholamines. J. Pharmacol. Exp. Ther. 138, 360?379 (1962)

2. Aoi, W., Henry, D. P., Weinberger, M. H.: Evidence for a physiological role of renal sympathetic nerves in adrenergic stimulation of renin release in the rat. Circ. Res. 38, 123?126 (1976)

3. Aoki, K., Takikawa, K., Hotta, K.: Role of adrenal cortex and medulla in hypertension. Nature, New Biol. 241, 122?123 (1973)

4. Baer, L., Knowlton, A., Laragh, J. H.: The role of sodium balance and the pituitary-adrenal axis in the hypertension of spontaneously hypertensive rats. In: Spontaneous hypertension (K. Okamoto, ed.), pp. 203?213. Tokyo: Igaku Shoin Ltd., 1972

5. Black, I. B., Hendry, I. A., Iversen, L. L.: Differences in the regulation of tyrosine hydroxylase and Dopa decarboxylase in sympathetic ganglia and adrenal. Nature, New Biol. 231, 27?29 (1971)

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