Congenital hypophosphataemia in adults: determinants of bone turnover markers and amelioration of renal phosphate wasting following total parathyroidectomy-Reference-Cited by-同舟云学术

Congenital hypophosphataemia in adults: determinants of bone turnover markers and amelioration of renal phosphate wasting following total parathyroidectomy

Published:2018-09-20 Issue:4 Volume:37 Page:685-693
ISSN:0914-8779
Container-title:Journal of Bone and Mineral Metabolism
language:en
Short-container-title:J Bone Miner Metab

Author:

McKenna Malachi J.,Martin-Grace Julie,Crowley Rachel,Twomey Patrick J.,Kilbane Mark T.

Publisher

Springer Science and Business Media LLC

Subject

Endocrinology,Orthopedics and Sports Medicine,General Medicine,Endocrinology, Diabetes and Metabolism

Link

http://link.springer.com/content/pdf/10.1007/s00774-018-0957-5.pdf

Reference32 articles.

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2. Quarles LD (2012) Skeletal secretion of FGF-23 regulates phosphate and vitamin D metabolism. Nat Rev Endocrinol 8:276–286

3. Gattineni J, Bates C, Twombley K, Dwarakanath V, Robinson ML, Goetz R, Mohammadi M, Baum M (2009) FGF23 decreases renal NaPi-2a and NaPi-2c expression and induces hypophosphatemia in vivo predominantly via FGF receptor 1. Am J Physiol Ren Physiol 297:F282–F291

4. Bijvoet OL (1969) Relation of plasma phosphate concentration to renal tubular reabsorption of phosphate. Clin Sci 37:23–36

5. Payne RB (1998) Renal tubular reabsorption of phosphate (TmP/GFR): indications and interpretation. Ann Clin Biochem 35:201–206

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