Wogonin inhibits tight junction disruption via suppression of inflammatory response and phosphorylation of AKT/NF-κB and ERK1/2 in rhinovirus-infected human nasal epithelial cells-Reference-Cited by-同舟云学术

Wogonin inhibits tight junction disruption via suppression of inflammatory response and phosphorylation of AKT/NF-κB and ERK1/2 in rhinovirus-infected human nasal epithelial cells

Published:2022-02-02 Issue:3 Volume:71 Page:357-368
ISSN:1023-3830
Container-title:Inflammation Research
language:en
Short-container-title:Inflamm. Res.

Author:

Kim Kyeong Ah,Jung Joo Hyun,Choi Yun Sook,Kim Seon Tae^ORCID

Funder

korea health industry development institute

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology,Immunology

Link

https://link.springer.com/content/pdf/10.1007/s00011-022-01542-w.pdf

Reference31 articles.

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2. Greenberg SB. Respiratory consequences of rhinovirus infection. Arch Intern Med. 2003;163:278–84.

3. Yuta A, Doyle WJ, Gaumond E, Ali M, Tamarkin L, Baraniuk JN, et al. Rhinovirus infection induces mucus hypersecretion. Am J Physiol. 1998;274:1017–23.

4. Wang JH, Kwon HJ, Jang YJ. Rhinovirus enhances various bacterial adhesions to nasal epithelial cells simultaneously. Laryngoscope. 2009;119:1406–11.

5. Yeo N, Jang YJ. Rhinovirus infection-induced alteration of tight junction and adherens junction components in human nasal epithelial cells. Laryngoscope. 2010;120:346–52.

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