Nilotinib-induced autophagic changes increase endogenous parkin level and ubiquitination, leading to amyloid clearance-Reference-Cited by-同舟云学术

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Nilotinib-induced autophagic changes increase endogenous parkin level and ubiquitination, leading to amyloid clearance

Published:2013-12-13 Issue:4 Volume:92 Page:373-386
ISSN:0946-2716
Container-title:Journal of Molecular Medicine
language:en
Short-container-title:J Mol Med

Author:

Lonskaya Irina,Hebron Michaeline L.,Desforges Nicole M.,Schachter Joel B.,Moussa Charbel E-H

Publisher

Springer Science and Business Media LLC

Subject

Genetics (clinical),Drug Discovery,Molecular Medicine

Link

http://link.springer.com/content/pdf/10.1007/s00109-013-1112-3.pdf

Reference46 articles.

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2. Greenfield JP, Tsai J, Gouras GK, Hai B, Thinakaran G, Checler F, Sisodia SS, Greengard P, Xu H (1999) Endoplasmic reticulum and trans-Golgi network generate distinct populations of Alzheimer beta-amyloid peptides. Proc Natl Acad Sci U S A 96:742–747

3. Skovronsky DM, Doms RW, Lee VM (1998) Detection of a novel intraneuronal pool of insoluble amyloid beta protein that accumulates with time in culture. J Cell Biol 141:1031–1039

4. Xu H, Sweeney D, Wang R, Thinakaran G, Lo AC, Sisodia SS, Greengard P, Gandy S (1997) Generation of Alzheimer beta-amyloid protein in the trans-Golgi network in the apparent absence of vesicle formation. Proc Natl Acad Sci U S A 94:3748–3752

5. Wang JY (2000) Regulation of cell death by the Abl tyrosine kinase. Oncogene 19:5643–5650

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