Transcriptome analysis revealed the mechanism of the metabolic toxicity and susceptibility of di-(2-ethylhexyl)phthalate on adolescent male ICR mice with type 2 diabetes mellitus-Reference-Cited by-同舟云学术

Transcriptome analysis revealed the mechanism of the metabolic toxicity and susceptibility of di-(2-ethylhexyl)phthalate on adolescent male ICR mice with type 2 diabetes mellitus

Published:2019-10-12 Issue:11 Volume:93 Page:3183-3206
ISSN:0340-5761
Container-title:Archives of Toxicology
language:en
Short-container-title:Arch Toxicol

Author:

Ding Yangyang,Gao Kun,Liu Yongchao,Mao Guanghua,Chen Kun,Qiu Xuchun,Zhao Ting,Yang Liuqing,Feng Weiwei,Wu Xiangyang

Funder

Specialized Research Fund for the Natural Science Foundation of China

China Postdoctoral Science Foundation

Postdoctoral Science Foundation of Jiangsu Province

Research Foundation for Senior Talents of Jiangsu University

Graduate Innovative Projects in Jiangsu Province

State Key Laboratory of Environmental Chemistry and Ecotoxicology Open Fund

Natural Science Foundation of Jiangsu Province

Collaborative Innovation Center for Water Treatment Technology and Materials

Publisher

Springer Science and Business Media LLC

Subject

Health, Toxicology and Mutagenesis,Toxicology,General Medicine

Link

http://link.springer.com/content/pdf/10.1007/s00204-019-02590-8.pdf

Reference71 articles.

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3. Amara I, Timoumi R, Annabi E, Neffati F, Najjar MF, Bouaziz C, Abid-Essefi S (2019) Di (2-ethylhexyl)phthalate induces cardiac disorders in BALB/c mice. Environ Sci Pollut Res 26(8):7540–7549

4. Amed S (2015) The future of treating youth-onset type 2 diabetes: focusing upstream and extending our influence into community environments. Curr Diabetes Rep 15(3):7

5. Asztalos BF, Schaefer EJ, Horvath KV et al (2007) Role of LCAT in HDL remodeling: investigation of LCAT deficiency states. J Lipid Res 48(3):592–599

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