The Ca2+-calmodulin-Ca2+/calmodulin-dependent protein kinase II signaling pathway is involved in oxidative stress-induced mitochondrial permeability transition and apoptosis in isolated rat hepatocytes-Reference-Cited by-同舟云学术

The Ca2+-calmodulin-Ca2+/calmodulin-dependent protein kinase II signaling pathway is involved in oxidative stress-induced mitochondrial permeability transition and apoptosis in isolated rat hepatocytes

Published:2014-03-11 Issue:9 Volume:88 Page:1695-1709
ISSN:0340-5761
Container-title:Archives of Toxicology
language:en
Short-container-title:Arch Toxicol

Author:

Toledo Flavia D.,Pérez Leonardo M.,Basiglio Cecilia L.,Ochoa Justina E.,Sanchez Pozzi Enrique J.,Roma Marcelo G.

Publisher

Springer Science and Business Media LLC

Subject

Health, Toxicology and Mutagenesis,Toxicology,General Medicine

Link

http://link.springer.com/content/pdf/10.1007/s00204-014-1219-5.pdf

Reference77 articles.

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3. Azarashvili T, Krestinina O, Odinokova I, Evtodienko Y, Reiser G (2003) Physiological Ca2+ level and Ca2+-induced permeability transition pore control protein phosphorylation in rat brain mitochondria. Cell Calcium 34:253–259. doi: 10.1016/S0143-4160(03)00107-6

4. Azzone GF, Azzi A (1965) Volume changes in liver mitochondria. Proc Natl Acad Sci USA 53:1084–1089. doi: 10.1073/pnas.53.5.1084

5. Baines CP, Song CX, Zheng YT, Wang GW, Zhang J et al (2003) Protein kinase Cepsilon interacts with and inhibits the permeability transition pore in cardiac mitochondria. Circ Res 92:873–880. doi: 10.1161/01.RES.0000069215.36389.8D

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