Heterocyclic amines reduce insulin-induced AKT phosphorylation and induce gluconeogenic gene expression in human hepatocytes
Author:
Funder
U.S. Public Health Service
Publisher
Springer Science and Business Media LLC
Subject
Health, Toxicology and Mutagenesis,Toxicology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s00204-023-03488-2.pdf
Reference61 articles.
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2. Alves TC, Befroy DE, Kibbey RG et al (2011) Regulation of hepatic fat and glucose oxidation in rats with lipid-induced hepatic insulin resistance. Hepatol Baltim Md 53:1175–1181. https://doi.org/10.1002/hep.24170
3. Brown MS, Goldstein JL (2008) Selective versus total insulin resistance: a pathogenic paradox. Cell Metab 7:95–96. https://doi.org/10.1016/j.cmet.2007.12.009
4. Carvalho AM, Miranda AM, Santos FA et al (2015) High intake of heterocyclic amines from meat is associated with oxidative stress. Br J Nutr 113:1301–1307. https://doi.org/10.1017/S0007114515000628
5. Cheng K-W, Chen F, Wang M (2006) Heterocyclic amines: chemistry and health. Mol Nutr Food Res 50:1150–1170. https://doi.org/10.1002/mnfr.200600086
Cited by 3 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Correction: Heterocyclic amines reduce insulin-induced AKT phosphorylation and induce gluconeogenic gene expression in human hepatocytes;Archives of Toxicology;2023-09-04
2. The effect of the rs1799931 G857A (G286E) polymorphism on N-acetyltransferase 2-mediated carcinogen metabolism and genotoxicity differs with heterocyclic amine exposure;Archives of Toxicology;2023-08-18
3. Induction of glucose production by heterocyclic amines is dependent on N-acetyltransferase 2 genetic polymorphism in cryopreserved human hepatocytes;Toxicology Letters;2023-07
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