Deletion of Both Rab-GTPase–Activating Proteins TBC1D1 and TBC1D4 in Mice Eliminates Insulin- and AICAR-Stimulated Glucose Transport-Reference-Cited by-同舟云学术

Deletion of Both Rab-GTPase–Activating Proteins TBC1D1 and TBC1D4 in Mice Eliminates Insulin- and AICAR-Stimulated Glucose Transport

Published:2014-09-23 Issue:3 Volume:64 Page:746-759
ISSN:0012-1797
Container-title:Diabetes
language:en
Short-container-title:Diabetes

Author:

Chadt Alexandra,Immisch Anja,de Wendt Christian,Springer Christian,Zhou Zhou,Stermann Torben,Holman Geoffrey D.,Loffing-Cueni Dominique,Loffing Johannes,Joost Hans-Georg,Al-Hasani Hadi

Publisher

American Diabetes Association

Subject

Endocrinology, Diabetes and Metabolism,Internal Medicine

Reference53 articles.

1. Regulation of glucose transport by insulin: traffic control of GLUT4

2. Subcellular localization and trafficking of the GLUT4 glucose transporter isoform in insulin-responsive cells

3. Insulin-stimulated Phosphorylation of a Rab GTPase-activating Protein Regulates GLUT4 Translocation

4. Substrate specificity and effect on GLUT4 translocation of the Rab GTPase-activating protein Tbc1d1

5. Distinct Signals Regulate AS160 Phosphorylation in Response to Insulin, AICAR, and Contraction in Mouse Skeletal Muscle

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