Valproic acid (VPA) exerts different effects on proliferation and viability of 4T1 parental and 4T1 cells overexpressing adaptor protein Ruk/CIN85 by triggering metabolic reprograming in type specific manner-Reference-Cited by-同舟云学术

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Valproic acid (VPA) exerts different effects on proliferation and viability of 4T1 parental and 4T1 cells overexpressing adaptor protein Ruk/CIN85 by triggering metabolic reprograming in type specific manner

Published:2024-09-10 Issue:3 Volume:40 Page:187-187
ISSN:0233-7657
Container-title:Biopolymers and Cell
language:
Short-container-title:Biopolym. Cell

Author:

Hudkova O. O.¹,Latyshko N. V.¹,Kishko T. O.¹,Krysiuk I. P.¹,Khudiakova O. V.¹,Skaterna T. D.¹,Horak I. R.¹,Drobot L. B.¹

Affiliation:

1. O.V. Palladin Institute of Biochemistry, NAS of Ukraine

Publisher

Institute of Molecular Biology and Genetics (NAS Ukraine)

Reference5 articles.

1. Overexpression of adaptor protein Ruk/CIN85 in mouse breast adenocarcinoma 4T1 cells induces an increased migration rate and invasion potential

2. Valproic acid suppresses Warburg effect and tumor progression in neuroblastoma

3. ADAPTOR PROTEIN Ruk/CIN85 PARTICIPATES IN THE METABOLIC CONTROL OF HUMAN BREAST ADENOCARCINOMA MCF-7 CELLS

4. Calix[4]arene chalcone amide C-1011 elicits differential effects on the viability of 4T1 mouse breast adenocarcinoma cells with different levels of adaptor protein Ruk/CIN85 expression

5. K-rasG12V transformation leads to mitochondrial dysfunction and a metabolic switch from oxidative phosphorylation to glycolysis

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