Adding ROS Quenchers to Cold K+Cardioplegia Reduces Superoxide Emission During 2-Hour Global Cold Cardiac Ischemia-Reference-Cited by-同舟云学术

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Adding ROS Quenchers to Cold K+Cardioplegia Reduces Superoxide Emission During 2-Hour Global Cold Cardiac Ischemia

Published:2011-01-31 Issue:1 Volume:17 Page:93-101
ISSN:1074-2484
Container-title:Journal of Cardiovascular Pharmacology and Therapeutics
language:en
Short-container-title:J Cardiovasc Pharmacol Ther

Author:

Aldakkak Mohammed¹,Stowe David F.¹²³⁴⁵,Heisner James S.¹,Riess Matthias L.¹²³⁴,Camara Amadou K. S.¹³

Affiliation:

1. Department of Anesthesiology, The Medical College of Wisconsin, Milwaukee, WI, USA

2. Department of Physiology, The Medical College of Wisconsin, Milwaukee, WI, USA

3. Cardiovascular Research Center, The Medical College of Wisconsin, Milwaukee, WI, USA

4. Zablocki Medical Center, Department of Veterans Affairs, Milwaukee, WI, USA

5. Department of Biomedical Engineering, Marquette University, Milwaukee, WI, USA

Publisher

SAGE Publications

Subject

Pharmacology (medical),Cardiology and Cardiovascular Medicine,Pharmacology

Link

http://journals.sagepub.com/doi/pdf/10.1177/1074248410389815

Reference49 articles.

1. Reduced reactive O2 species formation and preserved mitochondrial NADH and [Ca2+] levels during short-term 17 °C ischemia in intact hearts

2. ROS scavenging before 27°C ischemia protects hearts and reduces mitochondrial ROS, Ca2+ overload, and changes in redox state

3. Hypothermia augments reactive oxygen species detected in the guinea pig isolated perfused heart

4. N-acetylcysteine prevents reactive oxygen species–mediated myocardial stress in patients undergoing cardiac surgery: results of a randomized, double-blind, placebo-controlled clinical trial

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1. NADPH oxidase overexpression and mitochondrial OxPhos impairment are more profound in human hearts donated after circulatory death than brain death;American Journal of Physiology-Heart and Circulatory Physiology;2024-03-01

2. Hypothermia Prevents Cardiac Dysfunction during Acute Ischemia Reperfusion by Maintaining Mitochondrial Bioenergetics and by Promoting Hexokinase II Binding to Mitochondria;Oxidative Medicine and Cellular Longevity;2022-07-13

3. Myocardial protection by propolis during prolonged hypothermic preservation;Cryobiology;2019-06

4. Isoquercetin ameliorates myocardial infarction through anti-inflammation and anti-apoptosis factor and regulating TLR4-NF-κB signal pathway;Molecular Medicine Reports;2018-03-09

5. New Pharmacological Approaches to the Prevention of Myocardial Ischemia- Reperfusion Injury;Current Drug Targets;2017-10-18

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