Pronethalol decreases RBPJκ to reduce Sox2 in cerebral arteriovenous malformation
Author:
Affiliation:
1. Division of Cardiology, David Geffen School of Medicine at University of California Los Angeles (UCLA), Los Angeles, CA, USA
2. The Molecular Biology Institute at UCLA, Los Angeles, CA, USA
Publisher
SAGE Publications
Subject
Cardiology and Cardiovascular Medicine
Link
http://journals.sagepub.com/doi/pdf/10.1177/1358863X20942833
Reference12 articles.
1. Arteriovenous Malformations of the Brain
2. Reducing Jagged 1 and 2 levels prevents cerebral arteriovenous malformations in matrix Gla protein deficiency
3. Endothelial Notch4 signaling induces hallmarks of brain arteriovenous malformations in mice
4. Endothelial expression of constitutively active Notch4 elicits reversible arteriovenous malformations in adult mice
5. Elevated endothelial Sox2 causes lumen disruption and cerebral arteriovenous malformations
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1. Methylome analysis of endothelial cells suggests new insights on sporadic brain arteriovenous malformation;Heliyon;2024-08
2. Inhibition of endothelial histone deacetylase 2 shifts endothelial-mesenchymal transitions in cerebral arteriovenous malformation models;Journal of Clinical Investigation;2024-05-23
3. Self-restricted circular RNA circSOX2 suppressed the malignant progression in SOX2-amplified LUSC;Cell Death & Disease;2022-10-15
4. Pronethalol Reduces Sox2 (SRY [Sex-Determining Region Y]-Box 2) to Ameliorate Vascular Calcification;Arteriosclerosis, Thrombosis, and Vascular Biology;2021-02
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