Comprehensive and differential long-term characterization of the alpha-galactosidase A deficient mouse model of Fabry disease focusing on the sensory system and pain development-Reference-Cited by-同舟云学术

Comprehensive and differential long-term characterization of the alpha-galactosidase A deficient mouse model of Fabry disease focusing on the sensory system and pain development

Published:2016-01 Issue: Volume:12 Page:174480691664637
ISSN:1744-8069
Container-title:Molecular Pain
language:en
Short-container-title:Mol Pain

Author:

Üçeyler Nurcan¹,Biko Lydia¹,Hose Dorothea¹,Hofmann Lukas¹,Sommer Claudia¹

Affiliation:

1. Department of Neurology, University of Würzburg, Würzburg, Germany

Publisher

SAGE Publications

Subject

Anesthesiology and Pain Medicine,Cellular and Molecular Neuroscience,Molecular Medicine

Link

http://journals.sagepub.com/doi/pdf/10.1177/1744806916646379

Reference29 articles.

1. Fabry's disease

2. Pathological Study of the Sural Nerve in Fabry’s Disease

3. Involvement of Peripheral Nerve and Muscle in Fabry's Disease

4. Characterization of Pain in Fabry Disease

5. -Galactosidase A deficient mice: A model of Fabry disease

Cited by 24 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Restoration of peripheral neuropathy in Fabry mice via intrathecal administration of an adeno-associated virus vector encoding mGLA cDNA;Molecular Genetics and Metabolism;2024-09

2. Age-dependent small fiber neuropathy: Mechanistic insights from animal models;Experimental Neurology;2024-07

3. Fabry disease Schwann cells release p11 to induce sensory neuron hyperactivity;JCI Insight;2024-03-07

4. Small fibre neuropathy in Fabry disease: a human-derived neuronal in vitro disease model and pilot data;Brain Communications;2024

5. Endothelial Cell Dysfunction and Hypoxia as Potential Mediators of Pain in Fabry Disease: A Human-Murine Translational Approach;International Journal of Molecular Sciences;2023-10-21